Curcugen - 60 V-Caps - Genestra
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Or, Shop our Latest Deals:
Shop Now >- 300 mg
- Turmeric (Curcuma longa) Root Std. Extract (95% Curcumin)
- 150 mg
- Green Tea (Camellia sinensis) Leaf Std. Extract (25% Catechins)
Hypromellose, cellulose, stearic acid
Format
Veggie Caps
60 V-caps
Dosage
Take one capsule two times daily with meals or as recommended by your healthcare practitioner. For use beyond 12 weeks, consult your healthcare practitioner.
Important Information
Guaranteed to contain no added wheat, starch, yeast, gluten, corn, soy, sodium, sugar, artificial colouring or flavouring, antimicrobial preservatives, dairy or animal products. Ideal for vegans.
If you are pregnant or if you have a bile duct obstruction, do not use. If you are breastfeeding; if you are taking antiplatelet medication or blood thinners; or if you have gallstones, iron deficiency, stomach ulcers, excess stomach acid or a liver disorder, consult your healthcare practitioner prior to use. If you develop symptoms of liver trouble, consult your healthcare practitioner
- A good source of antioxidnts
- Ideal for vegans
- Anti-inflammatory
- Combination of turmeric and green tea extracts
- Antiviral and anticarcinogenic properties
Related Videos
No Related VideosArticles by a naturopathic doctor.
Genestra delivers professional-grade products that are offered in a variety of formats. Genestra natural health products have been proven safe, effective and reliable for over 30 years, and are backed by clinical evidence. Genestra brings you HMF Forte, HMF Immune, HMF UTI, Digest Plus, Probiotics, D-Mulsion and Super EFA Liquid.
Turmeric contains potent antioxidant chemicals, known as curcuminoids. They assist the body in neutralizing free radicals and help protect the liver from harmful toxins.
These same molecules have also proven to inhibit inflammation, thus acting as pain relieving agents. Turmeric has been shown to improve joint mobility and reduce the pain and swelling associated with all forms of arthritis. This activity is equivalent to the effect of some NSAID drugs.
Turmeric also lowers the level of harmful, or LDL, cholesterol and increases the level of helpful, or HDL, cholesterol. As well as improves circulation and blood flow. All of these properties make turmeric a good choice for individuals with compromised circulation, heart disease and high cholesterol.
Reducing Joint Pain Naturally
Most people over age 35 have at least some wear and tear in their joints, but that doesn't mean it should cramp your style.
Osteoarthritis is the most common type of arthritis. It usually has a gradual onset and begins after the age of forty. The knees, hips, fingers and vertebrae are most susceptible. The joint deterioration is due to faulty or deficient repair of bone and cartilage in the joint. The standard North American diet is deficient in the minerals necessary for proper joint repair; therefore it is not surprising that osteoarthritis is a common concern. Osteoarthritis causes stiffness and joint pain on motion.
It is typically worse in the morning. Initially there is no inflammation or swelling of the joint. As the damage progresses the joint may become inflamed and deformed. Muscles around the joint can tense up, joint mobility may decrease and there may be a grating feeling on movement. The pain becomes constant and occurs even when at rest.
Drug Treatments Actually Worsen Arthritis
Conventional treatment with non-steroidal anti-inflammatories has been shown to worsen arthritis. Side effects of some drugs are gastrointestinal bleeding, stomach ulcers, heart attacks and liver problems. Some of these medications, especially the NSAID group can actually accelerate the degradation of joint tissue. Corticosteroids like cortisone and prednisone sometimes recommended for arthritis, while effective at relieving discomfort in the short term, can cause a loss of bone mass.
Antioxidant
Numerous studies have shown that the various constituents of curcuma longa posses potent antioxidant properties. The ability of cucuminoids to reduce hydroxyl and peroxyl free radicals is well documented. Sharma reported curcumin to be an effective agent against lipid peroxidation.
Anti-inflammatory
The Central Drug Research Institute in India found curcumin to be the major constituent responsible for the anti-inflammatory activity. The classical model for studying acute effects of anti-inflammatory agents is to test their inhibitory action on the development of rat paw edema - the exudative phase of inflammation - induced, for instance, by the local injection of carrageenins. Thus inflammation is thought to be in part due to the action of prostaglandin derivative from arachidonic acid metabolism. A detailed evaluation of curcumin as a potential non-steroidal anti-inflammatory agent by Srimal and Dhawan found curcumin to be highly effective after oral administration. Curcumin was effective in other models of inflammation including granuloma, pouch, cotton pellet, formalin-induced, and Freund's adjuvant.
The mechanism of anti-inflammatory action of curcumin is not yet known. Some researchers found curcumin to be less effective in adrenalectomized rats, suggesting a participation of corticoidal steroids, while others did not observe any effect of curcumin salts on steroid release from the adrenal cortex. Recently, another, more specific in-vitro method has been developed which allows the study of the inhibitory mechanism of potential drugs. By using rat peritoneal neutrophilis, curcumin was tested for the direct effect on the 5-lipooxygenase activities. Another study found that curcumin was able to inhibit the production of pro-inflammatory mediators in microglial cells that had been stimulated to mount an inflammatory response. Microglial cells are activated after brain injuries and produce proinflammatory mediators and neurotoxic compounds. Curcumin decreased the production of these compounds, apparently by blocking NF-kB, a protein signal in the pathway that leads to their production. The overexpression of pro-inflammatory cytokines contributes to diseases such as Alzheimer's, cerebral ischemia and multiple sclerosis. The ability of curcumin to decrease inflammation presents an approach to slow the progression of these diseases.
Gastro-intestinal effects
Curcumin increases mucin content, thereby protecting the gastric mucosa against irritants. Controversial data exist regarding an anti-ulcerogenic activity of curcumin. Some researchers found a protective effect of curcumin against histamine-induced gastric ulceration, while others reported an ulcerogenic effect of curcumin.
Curcumin also possesses anti-spasmodic properties. Curcumin showed liver protective effects against carbon tetrachloride, D-Galactosamine and peroxide induced cytotoxicity. Curcumin increased bile acid production in dogs and rats.
Cardiovascular effects
A sharp and transient hypotensive effect of curcumin was reported in dogs. Curcumin also inhibited collagen and adrenaline-induced aggregation of platelets but did not affect prostacyclin (PGI2) synthesis.
Lipid metabolism
Rao and co-workers reported that rats fed with curcumin and cholesterol in their diet had only half to one-third of the serum and liver cholesterol levels compared to the controlled groups receiving cholesterol alone.
Anti-bacterial/Anti-fungal
Curcumin inhibited growth of most organisms including: Staph aureus, Streptococci, lactobacilli, corynebacterium, Baccilus aureus, and micrococcus pyogenes. The crude ether and chloroform extracts of curcuma longa showed fungistatic activity against several dermatophytes as well as anti-amoebic activity against Entamoeba histolytica.
Anti-viral
A 1993 study showed curcumin as an effective ally in the treatment against HIV. Curcumin was effective in inhibiting the replication of HIV in both acutely infected and chronically infected cells.
Anti-tumor activity
The anti-tumor activity of various extracts of curcuma longa has been remarked by several researchers. Topical application of curcumin inhibited the number of TPA-induced tumors by as much as 98%!! Curcumin was found to be a selective and non-competitive inhibitor of phosphorylase kinase.
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